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The erectile deficit secondary to bnsrrp is due to transection of the accessory pudendal arteries which act as the main cavernous arteries or is due to incomplete safeguarding of the nerves
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use within 30 days for optimal potency [9]
We cannot exclude the possibility that loss of fluorescence during AITC treatment was due to its interference with the biomarker, but it has been reported that exogenously supplied chemicals responsible for GSH depletion are not likely to interfere with roGFP2 52
Bae KY, Kim SW, Shin HY, et al
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